Aevita terrestrialis
100 nanometers
A. servo infecting Pgengi, Suchovenator terrestrecancer, in zone J1 were carried into zone J. There A. servo was able to infect many organisms in zone J. As it adapted to the variety of host physiologies A. terrestrialis arose.
They incorporate their host’s DNA into their DNA and vice versa, the effects are mostly negligible. Depending on the DNA they encounter, the parasites gene wills express differently. For unicellular organisms such such as Pyrrhocytos chomos and Nanocalvus petramcella they act like their ancestor, simply hijacking the host cell and making it produce more A. terrestrialis until the host bursts.
For species like S. cancovorem, they cause slight damage, mostly to the mucous membranes, due to the ease of access to cells, causing a mild runny-nose and tearing eyes. In more severe cases hosts can be left with blindness and respiratory difficulty.
In Terracrabbids like Terracrabbus killacrabus, the host instead foams at the mouth. This can be fatal if the Terracrabbid has poor access to water.
In Muscal hosts, infection ends when A. terrestrialis are pushed out by the fluids produced by their hosts as they fail to drift into more cells to infect. If the host fails to push them out it can lead to some damage of soft tissues.
In floral species, such as Fungradi cartius and Thermolongus homocaulis, the disease causes blemishing and cell death, the hosts typically kill tissue that is infected to prevent further spread.
A. terrestrialis last for around 21 days outside of a host. They are also marginally more resistant to temperature flux due to a hardier protein shell that doesn’t denature as easily. Generally they tend to die off outside of the host in more extreme seasons so infections peak during wet seasons. A. terrestrialis is capable of reinfecting hosts because many host immune systems lack the ability to recognize pathogens. It enters host cells by binding to protein receptors on the cell surface.
